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  • Therapeutische gebieden

Beyfortus® ▼(nirsevimab): effectiveness and public health impact in the real world

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Gaucher disease in children

Gaucher disease is an autosomal recessive lysosomal storage disorder caused by pathogenic variants in the GBA1 gene, resulting in deficient activity of the enzyme acid β glucosidase.

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Real-World Evidence and Phase IV studies in CRSwNP

Patients with chronic rhinosinusitis with nasal polyps (CRSwNP) generally have a high symptom burden and poor health-related quality of life (QoL), often requiring recurring systemic corticosteroid use and repeated sinus surgery.

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DUPIREAL: Two-Year Turning Point with Dupilumab in CRSwNP

The results from the DUPIREAL real-world study reinforce the efficacy of dupilumab demonstrated in the SINUS-24 and SINUS-52 randomized clinical trials and are consistent with the approved SmPC.1,2,3,4 DUPIREAL is the largest long-term real-world study (n=926) evaluating the effectiveness of dupilumab, including treatment response, disease control and remission, in severe uncontrolled CRSwNP over 2 years.2,3

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Efficacy and safety of dupilumab in patients with PN with or without a history of atopic comorbidities

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Breaking the Barrier: How to Manage Hypoglycaemia Fear in Diabetes Care

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Reframing the Insulin Conversation: Helping People With Type 2 Diabetes to Embrace Life

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FLUNITY-HD : the largest individually randomised influenza vaccine effectiveness study of its kind.

FLUNITY-HD is the largest individually randomised influenza vaccine effectiveness study, with nearly half a million older adults across multiple seasons.1

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Cumulative Life Course Impairment (CLCI) in Atopic Dermatitis

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Deliver-T (RWE Study): New Findings on Toujeo® for T2D Management Following Dual GIP/GLP-1 RA Therapy

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IL-4 and IL-13: centrale cytokines die type 2-inflammatie aansturen

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Understanding Gaucher Disease: Symptoms, Classification, and Clinical Presentation

Gaucher disease is a lysosomal storage disorder caused by a pathogenic variant in the gene responsible for producing the enzyme acid β-glucosidase. More than 300 alleles carrying pathogenic variants have already been identified.1

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